Atherosclerosis is reversible and preventable by several nutrients. Linus Pauling, Nobel Laureate in Chemistry & Peace, proved conclusively before his death at age 94 that Vitamin C alone will halt the disease process. Add vitamin E as mixed tocopherols and you cannot fail.

Vitamin C can cause the regression (reduction) of atherosclerotic plaque in persons afflicted with Atherosclerosis through its action of  accelerating the removal of calcium and cholesterol from atherosclerotic plaque. Vitamin E acts in a similar way, treating and preventing atherosclerosis by protecting LDL cholesterol and the endothelium or cells that line blood vessels from oxidation.

The big news is about two products which have the capacity to safely lower cholesterol levels without any untoward side effect, unlike prescription drugs, and just as effectively. Another vitamin proven to prevent and assist in reversing atherosclerosis is Folic Acid discovered by Roger J. Williams.

Cholesstanol is the latest breakthrough in natural medicines to assist in lowering total cholesterol levels, improving serum lipid profiles and angina, and reducing the LDL-cholesterol:HDL-cholesterol (LDL:HDL) ratio. Sugar cane wax acohols are naturally dervived and have been clinically proven in the management of cholesterol. Combined with an effective soy bean extract to provide totally natural alternative for lowering cholesterol and triglyceride levels to promote general good health.

References:
10mg per day of SCWAs will produce siginificant improvements in LDL-cholesterol and HDL_cholesterol. Up to 28% reduction in LDL and a 29% increase in HDL respectively over a twelve month period. Castano g. etal. Angiology 2001;52(2):115-125

Recall that you cannot obtain any Metagenics product without the requisite Practitioner code number. Obtainable only after the Health Assessment.

Next we have Co Enzyme Q10 the heart protector par excellence. Proven to give an older person the heart recovery time of a young person. Coenzyme Q10 helps to prevent Atherosclerosis by preventing the oxidation of LDL cholesterol and importantly  the CoQ10:LDL cholesterol ratio is even more important in the prevention of Atherosclerosis than is the HDL:LDL Cholesterol ratio.

This miracle substance, which the body manufactures until we age around 40 years then slows down, has a beneficial effect in every cell in the body of which there are up to 100 trillion. For the cardiovascular system alone we list:

Coenzyme Q10 stabilizes heartbeat in arrhythmia patients. Improves blood circulation, assists the heart to function normally, even when blood clots are present. Improves cardiomyopathy by 90%. At least 62% of cardiovascular disease patients are deficient in Coenzyme Q10. It alleviates congestive heart failure and improves the condition of 91% of heart attack patients within 30 days.

Coenzyme Q10 lowers blood pressure in persons afflicted with hypertension - this is by normalizing the body's Sodium:Potassium ratio. Normal or low blood pressure remains unaffected. Mitral valve prolapse responds in up to 87% of patients at the correct dosage.
 

Coenzyme Q10 is the nutrient of choice for cardiovascular problems associated with:

High Altitudes
Clogged Arteries
When Blood has high Fat content
During Angina and
Where blood clots affect the flow of oxygenated blood to heart muscle. All proven by reliable scientific medical research.

If you have cardiovascular problems or are aged 40 years or more, can you afford not to supplement Coenzyme Q10?

Lipoic Acid complements Co Enzyme Q10 and much more see Lipoic Acid health benefits

Folic Acid helps to prevent Heart Attacks
Supplemental Folic Acid at dosages ranging from 400 - 5,000 mcg lowers elevated Homocysteine (a substance widely implicated in various Cardiovascular Diseases) levels [scientific research - humans: 5 mg of Folic Acid per day for 4 weeks caused significant reduction in plasma Homocysteine levels] [scientific research - humans: 650 mcg of Folic Acid per day reduced elevated Homocysteine levels by 42%].

Folic Acid also prevents Ischemic Heart Disease [epidemiological evidence: people with a low daily intake of Folic Acid have a 69% greater risk of Ischemic Heart Disease than persons who consume 400 mcg or more of Folic Acid per day].

And don't forget to avoid Margarine!

JUST A FEW PAPERS AS CONFIRMATION

Margarine & Polyunsaturated oils
In 1983, the writer authored a paper titled " The Cholesterol Controversy". This paper showed that animal studies over the previous 30 years or more concluded that the over-consumption of partially hydrogenated oils resulted in an increase in cancer and heart disease. In spite of this information being readily available the National Health & Medical Research Council promoted the consumption of margarines and polyunsaturated cooking oils whilst cautioning against the consumption of a range of foods said to contain cholesterol and therefore to cause heart disease. This information was almost instantly taken up and promulgated by the National Heart Foundation who promoted this way of life for more than 40 years. The same fraudulent information was broadcast by so-called health authorities throughout the western world. The results are too terrible to contemplate. Countless numbers of individuals have died of cancer and heart disease, when eating natural foods such as milk, butter and olive oil would doubtless have saved many lives. View the following:

Margarine is a killer!
Researchers at the Harvard Medical School have concluded that consumption of trans-fatty acids significantly increases the risk of heart attack. Trans-fatty acids are found mainly in margarine and shortening and are formed during the hydrogenation of vegetable oils. The researchers studied 748 men aged 43 to 85 years over a three year period. They found a direct correlation between total cholesterol and the intake of trans-fatty acids and also between low-density-lipoprotein cholesterol (LDL) and trans-fatty acid intake. Blood concentration of the "good" cholesterol, high-density-lipoprotein cholesterol (HDL), on the other hand, decreased as the consumption of trans-fatty acids went up. The data translates into a 27% increased heart attack risk for a man consuming 30 grams per day of margarine as compared to a man consuming 10 grams per day or less. The researchers also discovered that people having high cholesterol levels to begin with were more susceptible to the adverse effects of trans-fatty acids. Thus to attempt to lower one's cholesterol level by switching to margarine may be precisely the wrong thing to do.
American Journal of Clinical Nutrition, December 1992, pp. 1019-24

Women are very susceptible
Women using margarine on a regular basis are more likely to suffer a heart attack than are women using butter. Doctors at the Harvard Medical School have now confirmed what has long been suspected - that margarine consumption may cause heart disease. The doctors have studied the diets and disease patterns of over 85,000 nurses since 1980. They found that women consuming more than four teaspoons of margarine per day had a 66% greater risk of developing heart disease than did women who consumed less than one teaspoon per month. Regular consumption of cookies (2-3 per day) and white bread (2- 3 slices per day) containing partially hydrogenated vegetable oils was associated with a 43-55% increase in heart disease. Women who had changed from butter to margarine to lower their cholesterol and avoid heart disease had a 67% higher incidence of heart disease than had women who had not made this switch. Women who took multivitamins and beta-carotene lowered their risk of heart disease while butter consumption was found not to increase the risk of heart disease. The researchers believe that trans-fatty-acids formed in the hydrogenation of vegetable oils are responsible for the increased rates of heart disease observed. Trans-fatty-acids found in animal fats on the other hand, did not appear to increase the risk of heart disease. It is ironic that the U.S. fast food industry recently have changed from using beef tallow to using hydrogenated vegetable oils. The french fries sold by McDonald's and Burger King are now fried in oil containing 24-35% of disease-causing trans-fatty-acids.
Willett, Walter C., et al. Intake of trans fatty acids and risk of coronary heart disease among women. The Lancet, Vol. 341, March 6, 1993, pp. 581-85

Fish Oil benefits countered by margarine
Fish oil supplements containing EPA (eicosapentaenoic acid) have an anti-inflammatory effect and may benefit people suffering from rheumatoid arthritis and psoriasis. This beneficial effect is significantly reduced when the diet is high in linoleic acid. A seven week controlled experiment involving 30 male volunteers was recently completed in Australia. The participants were given 1.6 gram EPA and 0.32 gram DHA (docosahexaenoic acid) daily. Half the volunteers were kept on a diet high in linoleic acid by using margarine as a spread and polyunsaturated oils for cooking. The other half used butter and olive oil which are low in linoleic acid. The experiment clearly showed that the incorporation of fish oil is enhanced by a diet containing butter and fish oil. Margarine and polyunsaturated oils had an inhibiting effect and should therefore be excluded from the diet in order to obtain maximum benefit from fish oil.
The American Journal of Clinical Nutrition, February 1992, pp. 395- 99

What price margarine now!

    Better than fish oil?

    you be the judge!

BIOLOGICAL TREATMENT
Includes Metagenics Protocols
40:30:30 Zone friendly diet together with a low-sodium high-magnesium/potassium ratio and with minimal red meats, plenty of fish (minimum 3 times per week) especially those high in omega-3 fatty acids, and/or  vitamin D; mackeral, salmon, sardines, herring, cod, mullet, tuna, and trout, otherwise one to two Fish Oil Capsule (1000mg) three times daily after meals, or regular consumption of at least one tablespoon (15g) of Chia seeds, i.e. at least every second day.   Free E-Book "Functional Nutrition" .

email: functionmed@gmail.com

EXERCISE :Walking at first normal and gradually building up to a brisk walk of at least one hour per day four to seven days per week. Swimming, cycling are both beneficial, for mind relaxation and exercise other sports such as golf or tennis.

MIND CONTROL: Biofeedback teaching ultimate relaxation and mind/ body control is the ultimate, also yoga, transcendental meditation or any other form of relaxation and meditation will benefit. Recall that what is matter does not matter. What is mind is all there is.

Avoid: Mental emotional stressors, fatty meats and refined foods, coffee, tobacco, salt, sugar and overeating.

 References:

· Toh, H. T. Improved isolated heart contractility and mitochondrial oxidation after chronic treatment with Panax ginseng in rats. American Journal of Chinese Medicine. 22(3-4):275-284, 1994.
· Qin, L. M., et al. [Experimental study on the cardiotonic action of extract from Codonopsis pilosula (Franch.)Nannf.I).] Chung Kuo Chung Yao Tsa Chih. 19(4):238-240, 1994.
· Langsjoen, H., et al. Usefulness of coenzyme Q10 in clinical cardiology: a long-term study. Mol Aspects Med. 15(Supplement):S165-S175, 1994.
· Seyde, W. C., et al. Carotenoid compound crocetin improves oxygenation in hemorrhaged rats. J Cereb Blood Flow Metab. December 6, 1986, pages 703-707.
· Chipperfield, B., et al. Magnesium and the heart. American Heart Journal. 93:679, 1977.
· Bailey, L. E. The effect of orotic acid on excitation-contraction coupling in dystrophic hamster hearts. Recent Development in Cardiac Muscle Pharmacology. Shibata and Bailey (eds.). Tokyo: Igaku-Shoin, pp. 1-12, 1982.
· Dohohoe, J. A., et al. The action of orotic acid as a positive inotropic agent during the acture phase of myocardial hypertrophy. Australian and New Zealand Journal of Medicine. 4:542-548, 1974.
· Pshennikova, M. G., et al. The influence of folic acid and orotic acids and actinomycin on the contractile function of the myocardium in hyperfunction of the heart. Kardiologia. 6(4):54, 1966.
· Simonson, E., et al. New approach in treatment of cardiac decompensation in USSR. Am. Heart J. 86:117-123, 1973.
· Stampfer, M. J., et al. Vitamin E consumption and the risk of coronary disease in women., New England Journal of Medicine. 328:1444-1448, 1993.
 

Homocysteine and Folic Acid

· Arnadottir, M., et al. The effect of high-dose pyridoxine and folic acid supplementation on serum lipid and plasma homocysteine concentrations in dialysis patients. Clinical Nephrology. 40(4):236-240, 1993.

· Jacob, R. A., et al. Homocysteine increases as folate decreases in plasma of healthy men during short term dietary folate and methyl group restriction. Journal of Nutrition. 124:1072-1080, 1994.

· Landgren, F., et al. Plasma homocysteine in acute myocardial infarction: Homocysteine-lowering effect of folic acid. J Int Med
. 237:381-388, 1995.

· Malinow, M. L., et al. Reduction of plasma hyocyst(e)ine levels by breakfast cereal fortified with folic acid in patients with coronary heart disease. The New England Journal of Medicine. 338:1009-1015, 1998.

· Stampfer, M. J., et al. Can lowering homocysteine levels reduce cardiovascular risk? New England Journal of Medicine. 332(5):328-329, 1995.

· Tucker, K. L., et al. Folic acid fortification of the food supply: Potential benefits and risks for the elderly population. Journal of the American Medical Association. 276(23):1879, 1996.

· Ubbink J. B., et al. Hyperhomocysteinemia and the response to vitamin supplementation. Clin Investig. 71:993-998, 1993.

· Ubbink, J. B. Vitamin B12, vitamin B6, and folate nutritional status in men with hyperhomocysteinemia. American Journal of Clinical Nutrition. 57:47-53, 1993.

· Ubbink, J. B., et al. Vitamin requirements for the treatment of hyperhomo-cysteinemia in humans. Journal of Nutrition. 124:1927-33, 1994.

· Van den Berg M., et al. Combined vitamin B-6 plus folic acid therapy in young patients with arteriosclerosis and hyperhomocysteinemia. Journal Vascular Surgery. 20(6):933-940, 1994.

· Verhoef, P., et al. Homocysteine metabolism and risk of myocardial infarction: relation with vitamins B6, B12, and folate. American Journal of Epidemiology. 143:845-849, 1996.