and Including ADHD

No more common disruptive family condition exists in our society than Attention Deficit or Hyperactivity Disorder. Studies show; the majority, most commonly consume a diet high in simple sugars.

Like many other chronic illness conditions, these two disorders did not exist as such per se fifty years ago. Certainly a very few children suffered some measure of hyper-activity, often coupled with consequent hypoactivity due to the over-consumption of simple sugars and/or refined carbohydrates. In other words, the response to hyperinsulinism, a condition which untreated most often becomes insulin resistance; commonly known as diabetes mellitus. Pemoline and Ritalin are the most commonly prescribed medications for these errors in carbohydrate metabolism or food sensitivity disorders. These drugs of course most often, like most drugs, give symptomatic relief but the underlying causative factor may proceed unchecked. It seems to me that if one has a cellular, nutritional aberration the  best treatment is applied nutrition, which is in effect "cellular biochemistry".  It is worthwhile checking this out: http://ritalinsideeffects.net/
 

Attention Deficit Disorder is a syndrome characterized by inability to focus Attention and periods of Hyperactivity - commonly exhibited in children but also affecting adults. ADD is believed to occur as a result of insufficient development of the Corpus Callosum of the Brain - this component of the brain is responsible for the interhemispheric flow of information valence. Food allergies/sensitivities and heavy metal toxicity are believed to be significant contributors to ADD. Implementation of a Metabolic Detoxification, Bowel Repair and Liver regeneration may assist by restoration of beneficial gut-flora, correcting digestion, repairing gut permeability and improving live detoxification of xenobiotics

Implementation of the Insulin Zone & or Metabolic Typing Diet  is highly recommended.          

Supplementation of DMAE has shown to significantly benefit children affected with ADD. Supplemental doses of DMAE around 300-3000mg per day has been shown to be effective in most treatment programs. DMAE increases Attention Span, decreases Aggression, improves Learning ability and increases Intelligence in 70% of ADD patients. DMAE also exerts a powerful calming effect on students. In a double blind placebo controlled cross over study, students administered 1,200 mg of DMAE per day for 5 days, exhibited better control of anxious reactivity under exam conditions.A military study showed that Tyrosine can increase concentration in a prolonged work environment. Subjects were supplemented with Tyrosine and it was found that concentration time and quality was improved.

Supplemental, exogenous NADH improves concentration in humans. NADH enhances the production of energy and dopamine in the brain which improves the ability to focus on tasks.
Phosphatidylserine has a dynamic beneficial effect on ADD.

Conclusion: Neuroactive nutrients represent an exciting advancement in the treatment of difficult to manage neurological disorders and their associated behavioral abnormalities. The synergistic use of these nutrients has provided even greater clinical responsiveness than the individual compounds themselves. Enhancing the repair and function of damaged neurological tissues is now more a reality than ever before.

Neuro Pro (Innovate Therapies)
Neuro Pro contains a powerful spectrum of nutrients specifically designed for their positive effect on neurological tissue. The clinical efficacy of these neuroactive compounds represents the cutting edge of scientific research into the treatment of neurological disorders and their associated behavioral abnormalities.
Each capsule contains:
L Tyrosine    400mg
DMAE (Dimethylaminoethanol)    200mg
N Acetyl Carnitine    50mg
Phosphatidylserine    15mg
NADH (Nicotinamide Adenine Dinucleotide Hydrogen)    1mg

Dosage: Take one or two tablets daily before food, or as directed by your health care practitioner.
Synergistic formula: Phosphatidylserine, Proxan, N Acetyl Carnitine.
Conditions in which the above nutrients may assist:
ADD (ADHD)   
Alzheimer’s Disease
Depression   
 Learning and Memory impairment
Parkinson's Disease   
Neuropathies
Intelligence   
 Hangovers   
Insomnia   
 Stress

Contra-indications: Patients with Epilepsy should not use Neuro Pro unless they are monitored by a physician.
Functions for which the above nutrients may assist:
 

And now a word or more of hope:http://www.davincimethod.com/

And for what it is worth the writer scored  90% for ADD in 2009, many of us border on genius!

References:

· Coleman, N., et al. Deanol in the treatment of hyperactive children. Psychosomatics. 17:68-72, 1976.
· Knoble, M. 2-Dimethylaminoethanol in behavior problems of children. Science Medicine (Buenos Aries). 119:939-944, 1961.
· Lewis, J. A., et al. Deanol and methylphenidate in minimal brain dysfunction. Clin Pharmacol Ther. 17:534-540, 1975.
· Oettinger, L. The use of Deanol in the treatment of disorders of behavior in children. Journal of Pediatrics. 53:671-675, 1958.
· Pfeiffer, C. C., et al. Stimulant effect of 2-dimethyl-l-aminoethanol: possible precursor of brain acetylcholine. Science. 126:610-611, 1957.
· Neri, D. F., et al. The effects of tyrosine on cognitive performance during extended wakefulness. Aviat Space Environ Med. 66(4):313-319, 1995.
· Acworth, I. N., et al. Tyrosine: Effects on catecholamine release. Brain Res Bull. 21(3):474-477, 1988.
· Agharanya, J., C., et al. Changes in catecholamine excretion after short-term tyrosine ingestion in normally fed human subjects. American Journal of Clinical Nutrition. 34(1):82-87, 1981.
· Alonso, R., et al. Elevation of urinary catecholamines and their metabolites following tyrosine administration in humans. Biol Psychiatry. 17(7):781-790, 1981.
· Gibson, C. J., et al. Physiological control of brain norepinephrine synthesis by brain tyrosine concentration. Life Sciences. 22:1399-1406, 1978.
· Owasoyo, J. O., et al. Tyrosine and its potential use as a countermeasure to performance decrement in military sustained operations. Aviat Space Environ Med. 63:364-369, 1992.
· Lino, A., et al. Psycho functional changes in attention and learning under the action of L acetylcarnitine in 17 young subjects. A pilot study of its use in mental deterioration. Clinica Terapeutica. 140:569-573, 1992
· Ghirardi, O., et al. Long-term acetyl-L-carnitine preserves spatial learning in the senescent rat. Progress in Neuropsychopharmacology & Biological Psychiatry. 13(1-2):237-245, 1989.
· Ghirardi, O., et al. Active avoidance learning in old rats chronically treated with levocarnitine acetyl. Physiol Behav. 52(1):185-187, 1992.
· Lino, A., et al. Psycho functional changes in attention and learning under the action of L acetylcarnitine in 17 young subjects. A pilot study of its use in mental deterioration. Clinica Terapeutica. 140:569-573, 1992.
· Kohjimoto, Y., et al. Effects of acetyl-L-carnitine on the brain lipofuscin content and emotional behavior in aged rats. Japanese Journal of Pharmacology. 48:365-371, 1988.
· Valerio, C., et al. The effects of acetyl-L-carnitine on experimental models of learning and memory deficits in the old rat. Funct Neurol. 4(4):387-390, 1989.
 

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